Lidocaine

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Product
Lidocaine
Posting date : Nov 17, 2014
Membership
Free Member Scince Jun 19, 2014
FOB Price
negotiate
Min. Order Quantity
10g
Supply Abillity
5000kg/month
Port
HongKong
Payment Terms
T/T; Western Union; Money Gram; Bank transfer.
Package
Packing according to the arrangement of customer orders
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Contact
Candice He
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Product Detail
Company Info
 
Quick Detail
Place of Origin
China [CN]
Brand Name
Guangzhouquanao
Model Number
skype:Candice148722
HS-CODE
3818-00
Package & Delivery Lead Time
Package
Packing according to the arrangement of customer orders
Delivery Lead Time
The day after the payment
Detailed Description
Anti-painingAnesthetic Anodyne for  Lidocaine CAS: 137-58-6
 
http://www.steroidsguy.com  my akype:Candice148722
Quick Detail:
 
Lidocaine
CAS: 137-58-6
Assay: 99%
MF: C14H23N2O58
MW: 235.3447
Reference Standard: BP2002/USP28
Character: White crystalline powderUsage: It replaces procaine,being widely used in plastic and cosmetic operation in local infiltration anesthesia, the sodium  channel inhibition of the nerve cell membrane to block nerve excitability and conduction.
 
 
Description:
Mechanism of action: Lidocaine stabilizes the neuronal membrane by inhibiting the ionic fluxes required for the initiation and conduction of impulses, thereby effecting local anesthetic action.
Hemodynamics: Excessive blood levels may cause changes in cardiac output, total peripheral resistance, and mean arterial pressure. With central neural blockade these changes may be attributable to block of autonomic fibers, a direct depressant effect of the local anesthetic agent on various components of the cardiovascular system and/or the beta-adrenergic receptor stimulating action of epinephrine when present. The net effect is normally a modest hypotension when the recommended dosages are not exceeded.
 
Applications:
Lidocaine crosses the blood-brain and placental barriers, presumably by passive diffusion.
Lidocaine is metabolized rapidly by the liver, and metabolites and unchanged drug are excreted by the kidneys. Biotransformation includes oxidative N-dealkylation, ring hydroxylation, cleavage of the amide linkage, and conjugation. N-dealkylation, a major pathway of biotransformation, yields the metabolites monoethylglycinexylidide and glycinexylidide. The pharmacological/toxicological actions of these metabolites are similar to, but less potent than, those of Lidocaine. Approximately 90% of Lidocaine administered is excreted in the form of various metabolites, and less than 10% is excreted unchanged. The primary metabolite in urine is a conjugate of 4-hydroxy-2, 6-dimethylaniline.
 
 
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